Abstract
The human gastric pathogen Helicobacter pylori possesses an enormous genomic plasticity and diversity that facilitates host adaptation. Despite the ancient association with its human host, this ε-proteobacterium can cause gastritis, ulcers and gastric cancer. Here we focus on multiple aspects of the genome level biology, from population genomics to re-evaluating the genus definition.
Original language | English |
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Pages (from-to) | 737-744 |
Number of pages | 8 |
Journal | Research in Microbiology |
Volume | 158 |
Issue number | 10 |
DOIs | |
Publication status | Published - Dec 2007 |
Externally published | Yes |
ASJC Scopus Subject Areas
- Microbiology
- Molecular Biology
Keywords
- ε-Proteobacteria
- Campylobacter
- Helicobacter
- Nickel uptake and transport
- Pathogenicity island
- Phase variation
- Signal transduction networks
- Type IV secretion systems
- Wolinella